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Gout In Spanish – Symptoms Of Gout And Why Should Spanish Speakers Care About It?

The translation of gout in Spanish is "gota."

Gout is caused by an accumulation of uric acid in the blood. If you create too much uric acid or your kidneys do not filter out enough, it may pile up and form tiny pointed crystals in and around joints.

These crystals may make the joint painfully inflamed (red and swollen). Gout is a kind of arthritis. According to estimates, gout affects 1-2 persons in every 100 in the UK. The illness mainly affects males over 30 and women after menopause.

Gout is more frequent in males than in women. Gout may be highly painful and incapacitating, but there are therapies available to help reduce symptoms and prevent future outbreaks.

What Is The Main Cause Of Gout?

Gout is caused by hyperuricemia, a disease in which the body has an excess of uric acid. When your body breaks down purines, which are contained in your body and the foods you consume, it produces uric acid.

When there is an excess of uric acid in the body, uric acid crystals (monosodium urate) may form in the joints, fluids, and tissues. Gout is not usually caused by hyperuricemia, and hyperuricemia without gout symptoms does not need treatment.

The following factors increase your chances of developing hyperuricemia, which causes gout:

  • Being a man
  • Being overweight
  • Having some medical issues, such as heart failure with congestive edema, hypertension (high blood pressure), insulin sensitivity, metabolic syndrome, diabetes, and kidney dysfunction
  • Taking drugs such as diuretics (water pills).
  • Having a drink. Gout becomes more likely as alcohol use increases.
  • Consuming fructose-rich foods and beverages (a type of sugar).
  • A diet heavy in purines, which the body converts to uric acid. Red meat, organ meat, and several types of seafood, such as anchovies, sardines, mussels, scallops, trout, and tuna, are high in purine.

Gout In Spanish Population

Gout, the most frequent type of inflammatory arthritis today, is becoming pandemic in the United States. With the frequency of comorbid health concerns such as diabetes, high blood pressure, and obesity on the increase, Hispanic Americans are at an even higher risk of acquiring gout.

"The Hispanic population is not only rising in number, but it also continues to constitute a big and growing part of gout patients," stated N. Lawrence Edwards, M.D., Chairman and CEO of GUAES and Professor of Medicine, Rheumatology, and Clinical Immunology at the University of Florida.

"Because more than three out of every four Hispanic Americans speak some Spanish, we are thrilled to launch additional Spanish-language gout resources and aim to extend our offerings in the future."

Through Spanish-language patient resources, it is critical to promote gout knowledge and urge correct diagnosis and care among at-risk Hispanic populations.

Gout Symptoms

Gout symptoms nearly typically appear quickly and frequently at night. They are as follows:

  • Severe joint pain: Gout often affects the big toe. However, it may affect any joint. Ankles, knees, elbows, wrists, and fingers are all often afflicted joints. The pain is most likely severe in the first four to twelve hours after it starts.
  • Persistent discomfort: Some joint soreness may remain from a few days to a few weeks after the most acute pain has subsided. Later episodes are more likely to be more severe and impact more joints.
  • Redness and inflammation: The afflicted joint or joints swell, become painful, heated, and red.
  • Range of motion is restricted: You may generally be unable to move your joints as your gout advances.
A woman's hands joined together
A woman's hands joined together

Gout Diagnosis

Gout is often diagnosed based on your symptoms and the look of the afflicted joint. Gout diagnostic tests may include:

  • Joint fluid analysis: Your doctor may use a needle to drain fluid from your damaged joint. When the liquid is viewed under a microscope, urate crystals may be detected.
  • Blood test: A blood test to determine the amounts of uric acid in your blood may be recommended by your doctor. However, blood test findings might be deceptive. Some individuals have elevated uric acid levels but never develop gout. And other patients have gout symptoms but no abnormal amounts of uric acid in their blood.
  • Imaging using X-rays: Joint X-rays may help rule out other potential causes of joint inflammation.
  • Ultrasound: Sound waves are used in this technique to identify urate crystals in joints or tophi.
  • Computerized tomography with dual energies: This technique combines X-ray pictures acquired from several angles to see urate crystals in joints.

Gout Treatment

Gout treatments may be quite effective. Gout treatment consists of two major components: (i) treating a severe attack and (ii) remedies against future attacks.

Gout Attack Treatment

The treatment of an acute gout attack differs significantly from preventing future occurrences. A variety of treatments may be taken to assist relieve a gout episode.

One idea is that therapy for a gout attack should begin as soon as possible since prompt treatment is generally rewarded with rapid improvement. If a gout episode is allowed to persist more than a day or two before therapy begins, the response to treatment may be substantially delayed.

  • COX-2 inhibitors or nonsteroidal anti-inflammatory medicines (NSAIDs): include naproxen 500mg twice daily and indomethacin 25 thrice daily. Celecoxib 200mg twice a day is an example of a COX-2 inhibitor. Possible adverse effects include high blood pressure, ankle edema, stomach distress, and ulcers (long-term use may increase the risk of heart attack or stroke, but gout use is generally very short-term). If you have renal or liver issues, use with care.
  • Corticosteroids with anti-inflammatory properties: Prednisone 40mg first day, 30mg second day, 20mg third day, and 10mg fourth day are anti-inflammatory corticosteroids. Possible adverse effects include an increase in blood pressure, an increase in blood sugar, and mood disturbances. Short-term usage, like gout, is often tolerated far better than long-term use. If you have diabetes, use it with care.
  • Colchicine: High dosages of colchicine were formerly used to treat gout episodes. However, this caused diarrhea in a substantial proportion of patients. Lower dosages of colchicine have been demonstrated to be just as helpful as large doses for gout attacks and considerably better tolerated. Assuming no other medical concerns need a dosage adjustment, a patient would get two tablets of colchicine, 0.6mg each, as soon as a gout attack begins. They would then be given one more pill an hour later. In individuals with considerably reduced renal function, colchicine dosage must be modified. Colchicine interacts with several drugs, the most notable of which is clarithromycin (Biaxin®).
  • Steroid injections: For example, varying dosages are utilized based on the size of the joint, and several formulations are available. Possible adverse effects include: A local response to the injection may occur 1-2 percent of the time, and the joint might temporarily deteriorate the following day, necessitating cold treatment. A single local injection may briefly elevate blood sugar in people with diabetes.
  • Avoid walking: Although exercise is beneficial for weight loss and is typically safe for gout sufferers, it is best to avoid activity as much as possible if you have a gout flare in your toe, foot, ankle, or knee. Pounding on an inflamed gouty joint might make it worse. Other types of activity, such as upper-body exercises, are OK, but the gouty joint should be rested. Another incentive to treat gout flares fast is because beginning early frequently means the flare will be brief, allowing you to minimize your time off your feet.

Prevention Of Gout Attack

Medications available may reduce urate levels, inhibit the formation of new crystals, and dissolve the crystals in your joints. Urate lowering treatments are what they're termed (ULTs). Treatment with ULTs is usually initiated when a gout episode has subsided entirely.

A ULT has no set dosage; different persons need varying doses to achieve the desired blood urate level. The medications may take months or years to rid your body of urate crystals completely. However, once they are gone, you will no longer have gout attacks, tophi, or the danger of joint injury caused by gout.

ULTs are often life-long therapies requiring annual check-ups to monitor your urate levels. If your symptoms do not improve, see your doctor about your urate level, as you may need a greater dosage. Try not to miss or skip doses, particularly during the first year or two of therapy. This might cause your urate levels to fluctuate, triggering an attack.

  • Allopurinol: The most often used ULT is allopurinol. It is a highly successful medication for the majority of gout patients. It works by lowering the quantity of urate produced by your body. You will begin with a modest dosage of allopurinol, progressively increasing until you reach the optimal amount. It is less likely to cause an attack if the dosage is gradually increased. It also ensures you are on the lowest possible dosage to manage your gout. Allopurinol is broken down and eliminated from the body via the kidneys; therefore, it may not be appropriate if you have renal problems. Your doctor may start you on a very low dosage and gradually raise it, or they may propose that you take febuxostat instead. If you are presently suffering from gout or are allergic to allopurinol, you will not be given it.
  • Febuxostat: Febuxostat is a newer medicine that, like allopurinol, decreases the quantity of urate produced in the body. Unless your doctor says you can't take allopurinol, you won't be given febuxostat as your first ULT. It functions similarly to allopurinol, except rather than being broken down by the kidneys, it is broken down by the liver. It is beneficial if you have renal difficulties and cannot take a high enough dosage of allopurinol. When you initially begin therapy, febuxostat is more likely than allopurinol to cause gout episodes. As a precaution, you will most likely be given a low-dose NSAID or colchicine to take for the first six months of febuxostat treatment. Because there are only two dosages of febuxostat, if your urate levels haven't decreased enough after a month on the low dose, you may need to switch to the higher dose.
  • Uricosuric drugs: Uricosuric medicines, such as sulfinpyrazone, benzbromarone, and probenecid, function by flushing your kidneys with more urate than usual. Because they are not readily accessible in the UK, they are seldom utilized. A rheumatologist will only prescribe them if allopurinol and febuxostat haven't worked or aren't appropriate for you. If you have severe renal issues or kidney stones, you will most likely be unable to take these medications. This is because uricosuric medicines urge your kidneys to filter more urate. This raises your chances of having kidney stones. Uricosuric medications are often administered on their own. However, in rare circumstances, if you've tried numerous ULTs and none have worked, uricosurics may be taken in conjunction with other ULTs such as allopurinol or febuxostat.
Visual description of human hand anatomy while rheumatoid arthritis is magnified
Visual description of human hand anatomy while rheumatoid arthritis is magnified

Food And Lifestyle

Medications are often the most effective treatment of gout episodes and avoid symptom flare-ups. However, lifestyle decisions are crucial as well, and you may wish to:

  • Select healthier drinks: Limit your intake of alcoholic beverages and drinks sweetened with fruit sugar (fructose). Instead, consume lots of nonalcoholic drinks, particularly water.
  • Purine-rich foods should be avoided: Purines are particularly abundant in red meat and organ meats such as the liver. Anchovies, sardines, mussels, scallops, trout, and tuna are examples of purine-rich seafood. Low-fat dairy products may be a superior source of protein for gout sufferers.
  • Lose weight by exercising regularly: Maintaining a healthy weight lowers your risk of gout. Low-impact exercises such as walking, biking, and swimming are better for your joints.

People Also Ask

What Is Gout And How Is It Treated?

Gout is an excruciatingly painful type of arthritis. When your body has an excess of uric acid, sharp crystals may develop in the big toe or other joints, producing swelling and agony known as gout attacks. Gout may be treated with drugs as well as dietary and lifestyle modifications.

What Are The 10 Foods That Trigger Gout?

Sugary beverages and sweets are among the top ten meals and drinks that cause gout.

  • High-fructose corn syrup
  • Alcohol
  • Organ meats
  • Game meats
  • Seafoods like herring, scallops, mussels, codfish, tuna, trout, and haddock
  • Red meats like beef, lamb, hog, and bacon
  • Turkey

How Do U Know If U Have Gout?

We have gout if:

  • The joint feels hot and sensitive to the point of being unable to stand anything touching it
  • Inflammation in and around the afflicted joint
  • The afflicted joint has red, glossy skin
  • As the edema subsides, the skin peels, becomes itchy, and flaky

Conclusion

Gout is a kind of arthritis that mainly affects the joints. It may cause excruciating pain, edema, and stiffness. More than 3 million Americans are affected by the illness, which is more common in men than women.

The most common cause of gout is hyperuricemia, or an excess of uric acid in the blood. Individuals may develop hyperuricemia if their bodies create too much uric acid or if their kidneys do not eliminate it effectively. To treat gout, a doctor will usually prescribe prescription drugs. This may include anti-inflammatory therapies and medications to assist manage uric acid levels. People may lower their chances of having gout by avoiding meals rich in purines, which the body turns into uric acid, staying hydrated, and avoiding alcohol.

About The Authors

Suleman Shah

Suleman Shah - Suleman Shah is a researcher and freelance writer. As a researcher, he has worked with MNS University of Agriculture, Multan (Pakistan) and Texas A & M University (USA). He regularly writes science articles and blogs for science news website immersse.com and open access publishers OA Publishing London and Scientific Times. He loves to keep himself updated on scientific developments and convert these developments into everyday language to update the readers about the developments in the scientific era. His primary research focus is Plant sciences, and he contributed to this field by publishing his research in scientific journals and presenting his work at many Conferences. Shah graduated from the University of Agriculture Faisalabad (Pakistan) and started his professional carrier with Jaffer Agro Services and later with the Agriculture Department of the Government of Pakistan. His research interest compelled and attracted him to proceed with his carrier in Plant sciences research. So, he started his Ph.D. in Soil Science at MNS University of Agriculture Multan (Pakistan). Later, he started working as a visiting scholar with Texas A&M University (USA). Shah’s experience with big Open Excess publishers like Springers, Frontiers, MDPI, etc., testified to his belief in Open Access as a barrier-removing mechanism between researchers and the readers of their research. Shah believes that Open Access is revolutionizing the publication process and benefitting research in all fields.

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